A controlled trial of reduced meal frequency without caloric restriction in healthy, normal-weight, middle-aged adults.
http://www.ncbi.nlm.nih.gov/pubmed/17413096
Normal-weight subjects are able to comply with a 1 meal/d diet. When meal frequency is decreased without a reduction in overall calorie intake, modest changes occur in body composition, some cardiovascular disease risk factors, and hematologic variables. Diurnal variations may affect outcomes.
Beneficial effects of intermittent fasting and caloric restriction on the cardiovascular and cerebrovascular systems.
http://www.ncbi.nlm.nih.gov/pubmed/15741046
Intermittent fasting (IF; reduced meal frequency) and caloric restriction (CR) extend lifespan and increase resistance to age-related diseases in rodents and monkeys and improve the health of overweight humans. Both IF and CR enhance cardiovascular and brain functions and improve several risk factors for coronary artery disease and stroke including a reduction in blood pressure and increased insulin sensitivity. Cardiovascular stress adaptation is improved and heart rate variability is increased in rodents maintained on an IF or a CR diet. Moreover, rodents maintained on an IF regimen exhibit increased resistance of heart and brain cells to ischemic injury in experimental models of myocardial infarction and stroke. The beneficial effects of IF and CR result from at least two mechanisms--reduced oxidative damage and increased cellular stress resistance. Recent findings suggest that some of the beneficial effects of IF on both the cardiovascular system and the brain are mediated by brain-derived neurotrophic factor signaling in the brain. Interestingly, cellular and molecular effects of IF and CR on the cardiovascular system and the brain are similar to those of regular physical exercise, suggesting shared mechanisms. A better understanding of the cellular and molecular mechanisms by which IF and CR affect the blood vessels and heart and brain cells will likely lead to novel preventative and therapeutic strategies for extending health span.
Caloric restriction and intermittent fasting: two potential diets for successful brain aging.
http://www.ncbi.nlm.nih.gov/pubmed/16899414
Intermittent fasting dissociates beneficial effects of dietary restriction on glucose metabolism and neuronal resistance to injury from calorie intake
http://www.pnas.org/content/100/10/6216.full
Intermittent fasting therefore has beneficial effects on glucose regulation and neuronal resistance to injury in these mice that are independent of caloric intake.
Effect of intermittent fasting and refeeding on insulin action in healthy men
http://jap.physiology.org/content/99/6/2128.full
This experiment is the first in humans to show that intermittent fasting increases insulin-mediated glucose uptake rates, and the findings are compatible with the thrifty gene concept.
[Physiological and biochemical effects of intermittent fasting combined with hunger-resistant food on mice].
http://www.ncbi.nlm.nih.gov/pubmed/17090373
The regimen of intermittent fasting combined with hunger-resistant food is safe and beneficial to metabolic regulation, such as controlling body-weight and adjusting blood glucose and serum lipid. It is expected that development of this regimen will be helpful to the control of obesity and diabetes, etc.
Intermittent fasting does not affect whole-body glucose, lipid, or protein metabolism
http://www.ajcn.org/content/90/5/1244.full
...+ kilkanascie badan na stronie leangains obalajacych mity typy wiecej poislkow = wiekszy TEF
6 poislkow lepiej niz 3
itd.
ogolnie z badan IF vs diety redukcyjne wychodzi remis - ew. z mala przewaga IF
"Cóż jest trucizną?
Wszystko jest trucizną i nic nie jest trucizną, tylko dawka czyni, że dana substancja nie jest trucizną!".
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