Pytanie na temat low carb

od pewnego momentu leptyna odgrywa zbyt wielka wage

Może tak, może nie...

Leptin and energy expenditure.

Hukshorn CJ, Saris WH.

Nutrition and Toxicology Research Institute Maastricht (NUTRIM), Department of Human Biology, Maastricht University, Maastricht, The Netherlands. [email protected]

PURPOSE OF REVIEW: A fundamental advance in our understanding of endocrine control of energy balance and body weight came with the discovery of the adipocyte-derived hormone leptin. The leptin pathway appeared to be the long-sought peripheral signal pathway from the adipose tissue to the brain involved in the regulation of feeding and energy balance. RECENT FINDINGS: Initially, leptin was considered to function as the long-sought antiobesity hormone. According to this hypothesis, rising concentrations of leptin with increasing adiposity would generate a signal to reduce food intake and increase energy expenditure in order to limit further weight gain. However, widespread resistance to the proposed antiobesity action of leptin is observed in humans, which might reflect the fact that the inability to store energy efficiently at times of abundance is evolutionarily disadvantageous. According to this alternative view, falling leptin concentrations observed during fasting act as a peripheral signal of starvation, which serves to conserve energy in the face of limited reserves. However, leptin administration failed to blunt the changes in energy expenditure during severe energy restrictions in several clinical studies. In addition, leptin therapy in several different human low-leptin states failed to affect energy expenditure in recent studies. SUMMARY: Increasing evidence from human studies suggests that leptin predominantly influences the human energy balance through appetite but appears not to be involved in regulating energy expenditure. None of the expected factors such as resting metabolic rate, total diurnal energy expenditure or dietary induced thermogenesis was related to blood leptin concentrations.

Tutaj jeszcze fragment opini Johna Berardi:

A: Lyle McDonald (more of a leptin expert than I’ll ever be) and I just had a discussion about this with David Greenwalt and his coaching group over at leannesslifestyle.com and we both pretty much concluded that refeeds probably won’t do much for dieters in terms of leptin. For starters, leptin kicks up and down very rapidly as energy intake fluctuates. Therefore, while leptin may kick up with a 10-hour carbohydrate reefed, it’s likely to drop back down just as rapidly after the reefed is over and another 10 hours of dieting are accomplished. Therefore, a dieter may just end up with a bigger positive energy balance during those 24hours of refeeding and subsequent return to dieting.

Since there is no data, one way or the other, illustrating what happens in dieting weight lifters when refeeding, there's only speculation. Of course, leptin itself aside, if there were some prolonged increase in leptin, we should be able to measure the effects of this leptin increase by observing increases in metabolic rate the day after the refeed. Unfortunately, metabolic increases as a result of acute overfeeding aren’t observed a day after the overfeed (or refeed). Remember, we’re not so much interested in what’s happening with leptin itself but what’s happening to metabolism. Leptin doesn’t impact fat loss. The effects of leptin do. And it doesn’t appear that refeeds impact metabolism for any longer than the day of the refeed.

However, make no mistake about it. I am not saying that refeeds are useless. In fact, I do see other good reasons (i.e. a psychological break from dieting, increased adherence, better glycogen status, more intense workouts, suppression of the catabolic hormonal cascade associated with dieting) for refeeding besides the leptin issue.

Zmieniony przez - ellis w dniu 2006-12-19 11:57:40

tylko ile bylo tych dni refeed ? lyle wspominal o minimum 2-3

Zmieniony przez - qazar w dniu 2006-12-19 13:43:38

Tak pisał:

There is also the issue of whether or not a short term bump (i.e. 5 hour refeed) is sufficient to have an imapct on metaboli rate.

In the UD2, I discussed this issue in some detail. In that it usually take 3-4 days of dieting for metabolism to decrase at all, I would be surprised if the body could raise metabolic rate in a ver short period of time. I vaguely recall a rat study where 1 day of overfeeding bumped metabolic rate but 1 day for a rat is 3-7 days for a human.

Which was at least part of why the UD2 used 3 days of overfeeding

mysle,ze jak juz ktos posiada zdolnosc czytania na tym poziomie w jezyku angielskim to sam sobie moze odnalezc te cytaty
ale gratuluje wszechstronnosci,wiele wnosza do dyskusji

jakbys ktos to przetlumaczyl to bym byl wdzieczny:>

Problem rozbja sie w naszych rozmowach o wplyw ladowan weglowdanami na poziom leptyny

wiadomo jest ze juz kolo 3-4 dnia redukcji nastepuja widoczne znaki obciecia kcal, poziom leptyny spada o pare-dziesat procent, co neistety nie wiaze sie ze spadkiem pare-dziesiat % tk. tluszczowej i teraz pytanie czy 3-4 dniowe ladowania weglowdnami moga odwrocic ten proces ? teoria jest mhmm zamglona w tej sprawie , praktyka pokazuje jednak ze da rade

z tego co sie zorientowalem to leptyna wystepuje w wiekszosci w warstwach podskornych,skad najtrusniej w prektyce pozbyc sie tkanki tluszczowej
proces "odtluszczania" ma swoj koniec chyba,ze wspomozemy go sroskami tarczycowymi i hormonem wzrostu co doprowadza do charakterystycznego "odrywania sie skory od miesnia" normalnie nie do osiagniecia w mlodym wieku,ale zauwazalnym u ludzi starszych - wiec moze wnioskujac z tego komorki te maja jakas (bo maja) bardzo obronna funkcje dla naszego organizmu.To juz moze porusza koledzy majacy wiele wiecej do powiedzenia na ten temat.

leptyna to hormon komunikujacy sie z mozgiem i mowiacy mu ile jemy i ile tluszczu posiadamy im mniej jemy i im mniej mamy tluszczu tym mniej leptyny dociera do mozgu tym ten bardziej chroni tluszcz bo wie ze wlasnei zaczela sie smierc glodowa

https://www.sfd.pl/LEPTYNA_-_hormon_tkanki_tłuszczowej-t122369.html

Zmieniony przez - qazar w dniu 2006-12-19 17:28:56

popieram postulat future by ellis przestal dawac cytaty w jezyku ktorego nie rozumiem ! co do tego oczym pisales lukaszu to znam osobiscie pakera ktory osiagnal tzw odrywanie sie skory od miesni - wlasnie na ckd - podobno poziom tkanki tl mial okolo 4% /mowa o koledze P.P./
Faktem jest tez to ze tkanka tluszczowa pelni tak wielwe funkcji /rownierz i hormonalne/ ze calkowite pozbycie sie jej na dluzsza mete zagraza zdrowiu, a nawet zyciu.
pozdr
mc

Zmieniony przez - marek.c w dniu 2006-12-19 17:42:00