od Lyle
As well, considering the generally small amount of calories/anything burned during activiyt, it's far more important to look at 24 hour energy/nutrient balance.
And the fact is that glycogen depleting (higher intensity) exercise tends to enhance 24 hour fat oxidation as incoming carbs are used to refill glycogen instead of for energy (abstract below). High intensity exercise also
a. burns more claories/unit time during the workout
b. increases EPOC far moreso than lower intensity activity
So, from a fat loss standpoint, you have to ask which is more important:
burning more fat over the hour you do cardio?
burning more fat over the OTHER 23 hours of the day?
Lyle
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J Physiol. 2003 May 1;548(Pt 3):919-27. Epub 2003 Mar 21.
Skeletal muscle fat and carbohydrate metabolism during recovery from
glycogen-depleting exercise in humans.
Kimber NE, Heigenhauser GJ, Spriet LL, Dyck DJ.
Department of Human Biology and Nutritional Sciences, University of Guelph,
Guelph, Ontario N1G 2W1.
The primary aim of the present study was to determine whether intramuscular
triacylglycerol (IMTG) utilization contributed significantly to the increase in
lipid oxidation during recovery from exercise, as determined from the muscle
biopsy technique. In addition, we also examined the regulation of pyruvate
dehydrogenase (PDHa) and changes in muscle acetyl units during an 18 h recovery
period after glycogen-depleting exercise. Eight endurance-trained males
completed an exhaustive bout of exercise (~90 min) on a cycle ergometer followed
by ingestion of carbohydrate (CHO)-rich meals (64-70 % of energy from
carbohydrate) at 1, 4 and 7 h of recovery. Duplicate muscle biopsies were
obtained at exhaustion, and 3, 6 and 18 h of recovery. Despite the large intake
of CHO during recovery (491 +/- 28 g or 6.8 +/- 0.3
g kg-1), respiratory
exchange ratio values of 0.77 to 0.84 indicated a greater reliance on lipid as
an oxidative fuel. However, there was no net IMTG utilization during recovery.
IMTG content at exhaustion was 23.5 +/- 3.5 mmol (kg dry wt)-1, and remained
constant at 24.6 +/- 2.6, 25.7 +/- 2.8 and 28.4 +/- 3.0 mmol (kg dry wt)-1 after
3, 6 and 18 h of recovery. Muscle glycogen increased significantly from 37 +/-
11 mmol (kg dry wt)-1 at exhaustion, to 165 +/- 13, 250 +/- 18, and 424 +/- 22
mmol (kg dry wt)-1 at 3, 6 and 18 h of recovery, respectively. PDHa was reduced
at 6 and 18 h when compared to exhaustion, but did not change during the
recovery period. Acetyl-CoA, acetylcarnitine and pyruvate contents declined
significantly after 3 h of recovery compared to exhaustion, and thereafter
remained unchanged. We conclude that IMTG has a negligible role in contributing
to the enhanced fat oxidation during recovery from exhaustive exercise. Despite
the elevation of glucose and insulin following high-CHO meals during recovery,
CHO oxidation and PDH activation were decreased, supporting the hypothesis that
glycogen resynthesis is of high metabolic priority. Plasma fatty acids, very low
density lipoprotein triacylglycerols, as well as intramuscular acetylcarnitine
stores are likely to be important fuel sources for aerobic energy, particularly
during the first few hours of recovery.
Fakty, nie mity - stan wiedzy na rok 2003
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