badania na temat wplywu sterydow w dawkach jakie my przyjmujemy mozna policzyc na palacach (glownie leczenie aids poparzen itp) wiec raczej tego nie znajdziesz.
Biologia jest jednak nieublagana androgen z zewn = zmniejszenie wydzielania lh fsh. Jesli bys bral jakis srodek malo androgenny solo a nie po cyklu i do tego dosyc krotko i w malej dawce to wtedy mozna mowic ze cos "malo" blokuje. Lh i FSH na 100% by sie obnizylo ale nie bylby to blok totalny lub bliski zeru.
Ty bedziesz po cyklu a to calkowicie zmienia postac rzeczy. Zrob odblok jak nalezy bez zadnego koxu bo to dla mnie totalne nieporozumienie. Albo PCT albo jedziesz dalej z cyklem (mostki itp) ale to zabawa dla profesjonalistow imho.
Ps jak chcesz badania to masz .......dawka oxy 2.5 mg powoduje zmniejszenie wydzielania hormonow przysadki
1: Clin Endocrinol (Oxf) 1997 Feb;46(2):209-16 Related Articles, Books, LinkOut
Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.
Crowne EC, Wallace WH, Moore C, Mitchell R, Robertson WH, Holly JM, Shalet SM.
Department of Endocrinology, Christie Hospital Trust, Manchester, UK.
OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes. DESIGN: Prospective double-blind placebo-controlled trial. PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months. MAIN OUTCOME MEASURES: LH and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. Testosterone levels were measured hourly and insulin, SHBG, IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures. RESULTS: LH and testosterone parameters increased significantly with time in all 16 (LH AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning
testosterone, P = 0.002). In Group 2, however, LH and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. SHBG levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months. CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. Testosterone increased GH via amplitude modulation.
Publication Types:
Clinical trial
Randomized controlled trial
Zmieniony przez - Roid_rage w dniu 2009-09-06 22:39:32