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np tutaj www.mybodycomp.com (trzeba sie logowac)
a great flame follows a little spark
Budujesz mięśnie - http://www.sfd.pl/Robimy_mase_-_fakty_nie_mity_by_qaz-t173140.html
Redukujesz tkankę tłuszczową - http://www.sfd.pl/10_wskazówek_żywieniowych-t315293.html
Chcesz być zdrowy ? - http://www.sfd.pl/Równowaga_kwasowo-zasadowa-t344643.html
http://www.sfd.pl/Zdrowie_bez_tluszczu_nie_mozliwe!!!-t164189.html#post0
Effect of caloric restriction and dietary composition of serum T3 and reverse T3 in man.
Spaulding SW, Chopra IJ, Sherwin RS, Lyall SS.
To evaluate the effect of caloric restriction and dietary composition on circulating T3 and rT3 obese subjects were studied after 7-18 days of total fasting and while on randomized hypocaloric diets (800 kcal) in which carbohydrate content was varied to provide from 0 to 100% calories. As anticipated, total fasting resulted in a 53% reduction in serum T3 in association with reciprocal 58% increase in rT3. Subjects receiving the no-carbohydrate hypocaloric diets for two weeks demonstrated a similar 47% decline in serum T3 but there was no significant change in rT3 with time. In contrast, the same subjects receiving isocaloric diets containing at least 50 g of carbohydrate showed no significant changes in either T3 or rT3 concentration. The decline in serum T3 during the no-carbohydrate diet correlated significantly with blood glucose and ketones but there was no correlation with insulin or glucagon. We conclude that dietary carbohydrate is an important regulatory factor in T3 production in man. In contrast, rT3 concentration is not significantly affected by changes in dietary carbohydrate. Our data suggest that the rise in serum rT3 during starvation may be related to more severe caloric restriction than that caused by the 800 kcal diet.
Relationships between iodothyronine peripheral metabolism and ketone bodies during hypocaloric dietary manipulations.
Pasquali R, Baraldi G, Biso P, Pasqui F, Mattioli L, Capelli M, Calliva R, Spoto M, Melchionda N, Labo G.
Relationships between iodothyronine and metabolic substrate metabolism during undernutrition were evaluated in four normal subjects who fasted for 48h (Group I) and in four groups (II to V) of obese patients who underwent selective dietary manipulations: 360 calories [carbohydrate (CHO) 40 g/day]; 800 calories containing respectively 19 g/day - ketogenic - (K) and 112 g/day - non ketogenic - (NK) of CHO; and a step-diet programme (during which total calories were progressively reduced from 2500 to 500). Serum T3 levels decreased significantly and constantly during fasting, 360 and 800 K studies, and transiently during the 800 NK diet. During the step-diet programme, a significant fall was found only when 1250 K or less were given. Conversely, serum reverse T3 rose significantly and constantly during 360 and 800 K diets, while a transient increase was found during the 800 NK diet. During the step-diet programme reverse T3 rose only when 750 calories were given. Ketogenesis developed in all studies but one (800 NK), and in the step-diet programme significantly below the 1000 calorie step. Other substrate modifications in each study were also evaluated. Serum T3 levels showed a significant correlation with ketone bodies (KB) in all the ketogenic studies, while no correlation was found in non ketogenic study (800 NK). During the step-diet programme ketone bodies and iodothyronine modifications appeared to be related to the amount of calories. Based on these results, we suggest a relationship between the dietary-induced modifications of iodothyronine metabolism and the development of ketogenesis.
The role of dietary fat in peripheral thyroid hormone metabolism.
Otten MH, Hennemann G, Docter R, Visser TJ.
Short term changes in serum 3,3',5-triiodothyronine (T3) and 3,3'5-triiodothyronine (reverse T3, rT3) were studied in four healthy nonobese male subjects under varying but isocaloric and weight maintaining conditions. The four 1500 kcal diets tested during 72 hr, consisted of: I, 100% fat; II, 50% fat, 50% protein; III, 50% fat, 50% carbohydrate (CHO), and IV, a mixed control diet. The decrease of T3 (50%) and increase of rT3 (123%) in the all-fat diet equalled changes noted in total starvation. In diet III (750 kcal fat, 750 kcal CHO) serum T3 decreased 24% (NS) and serum rT3 rose significantly 34% (p < 0.01). This change occurred in spite of the 750 kcal CHO. This amount of CHO by itself does not introduce changes in thyroid hormone levels and completely restores in refeeding models the alterations of T3 and rT3 after total starvation. The conclusion is drawn that under isocaloric conditions in man fat in high concentration itself may play an active role in inducing changes in peripheral thyroid hormone metabolism.
Effect of dietary carbohydrates during hypocaloric treatment of obesity on peripheral thyroid hormone metabolism.
Pasquali R, Parenti M, Mattioli L, Capelli M, Cavazzini G, Baraldi G, Sorrenti G, De Benedettis G, Biso P, Melchionda N.
The effect of different hypocaloric carbohydrate (CHO) intakes was evaluated in 8 groups of obese patients in order to assess the role of the CHO and the other dietary sources in modulating the peripheral thyroid hormone metabolism. These changes were independent of those of bw. Serum T3 concentrations appear to be more easily affected than those of reverse T3 by dietary manipulation and CHO content of the diet. A fall in T3 levels during the entire period of study with respect to the basal levels occurred only when the CHO of the diet was 120 g/day or less, independent of caloric intake (360, 645 or 1200 calories). Moreover, reverse T3 concentrations were found increased during the entire period of study when total CHO were very low (40 to 50 g/day) while they demonstrated only a transient increase when CHO were at least 105 g/day (with 645 or more total calories). Indeed, our data indicate that a threshold may exist in dietary CHO, independent of caloric intake, below which modifications occur in thyroid hormone concentrations. From these results it appears that the CHO content of the diet is more important than non-CHO sources in modulating peripheral thyroid hormone metabolism and that the influence of total calories is perhaps as pronounced as that of CHO when a "permissive" amount of CHO is ingested.
To ostatnio czytałem i mnie mocniej zaciekawiło, muszę jeszcze poczytać linki powiazane, bo to jest z 1977 r.
Alterations in basal and TRH-stimulated serum levels of thyrotropin, prolactin, and thyroid hormones in starved obese men.
Carlson HE, Drenick EJ, Chopra, IJ, Hershman JM.
To investigate further the alterations in pituitary-thyroid function seen during starvation, we have measured basal and TRH-stimulated serum levels of thyrotropin (TSH), prolactin (PRL), growth hormone, thyroxine (T4), triiodothyronine (T3), free T4, free T3, and reverse T3 during prolonged fasting in seven obese men. Fasting was associated with a significant decrease in serum (4, (3, and free T3, while there was an increase in serum reverse T3; these values tended to return toward pre-fast levels as the fast continued beyond 3 weeks. No significant changes were seen in basal serum TSH, PRL, growth hormone, or free T4. Although the TSH response to TRH was diminished during fasting, PRL, T4, and T3 responses were unchanged. In addition to transient alterations in the peripheral metabolism of T4, these findings suggest that alterations in the thyroid hormone binding capacity of serum carrier proteins may occur during fasting. The blunted TSH response to TRH despite reduction of serum T3 concentration suggests that subtle alterations in hypothalamic-pituitary function may also occur.
Masz jeszcze artykuł:
http://www.avantlabs.com/magmain.php?issueID=10&pageID=100
Badania w tym temacie mówią, że bezpieczna granica ilości węglowodanów to 50 g lub 120 g. Dodatkowo u osób z niedoczynnością tarczycy, gdy problem będzie się pogłębiał może być nieciekawie. Jeśli miałbym doradzić coś od siebie to nie mniej niż 50 g węglowodanów w diecie, a najlepiej 120 g i co jakieś 4-5 tygodni, redukuj deficyt do zera i podnoś ilość węglowodanów do 150 g na 1-2 tygodnie. Nie biorę odpowiedzialności za te porady na Twoim miejscu udałbym się do dobrego endokrynologa.
Zmieniony przez - ellis w dniu 2006-03-16 10:51:23
Jesli beda kiepskie zwieksze wegle do 150, jesli bedzie OK pozostane przy obecnej SKD i wlacze Twoje sugestie (okresowe zwiekszenie bilansu i ww).
Co do lekarza to z obecnego jestem zadowolona (z poprzednich 5 ! nie bylam) - niestety znalezienie endokrynologa ktory jednoczesnie zna sie na dietetyce graniczy z cudem.
Zmieniony przez - ellis w dniu 2006-03-16 19:07:35
1
"Prosze Pani, Pani ma niedoczynnosc przysadki. Czy ktos wczesniej robil Pani rezonans?"
Ja: "Nie"
"A to w takim razie nie ma potrzeby..."
2
"Uuuuuuuuu.... Pani ma niedoczynnosc tarczycy. No to wypiszemy leki (...). I jeszcze polece Pani wspaniale witaminki (tu pada nazwa). Tak sie sklada ze jestem ich dystrybutorem... Bez nich leczenie bedzie niekompletne. Tylko 150 zl miesiecznie...."
Itp.
Rece opadaja... Na nastepnej wizycie zapytam o wplyw diety nisko ww na tarczyce. To dopiero beda jaja...
Budujesz mięśnie - http://www.sfd.pl/Robimy_mase_-_fakty_nie_mity_by_qaz-t173140.html
Redukujesz tkankę tłuszczową - http://www.sfd.pl/10_wskazówek_żywieniowych-t315293.html
Chcesz być zdrowy ? - http://www.sfd.pl/Równowaga_kwasowo-zasadowa-t344643.html
http://www.sfd.pl/Zdrowie_bez_tluszczu_nie_mozliwe!!!-t164189.html#post0
aaa... i jakos nie jestem przekonany do duzych ladowan w stylu 10g ww/kg masy, znacznie chetniej stosowalbym te ladowanka nocne 100gram ww, pzdr
Zmieniony przez - Ritual w dniu 2006-03-16 21:38:28
Zmieniony przez - ellis w dniu 2006-03-16 22:29:00