omka

"....proviron imo nie na ten wiek.. po co jechac po prostacie i watrobie..."
Om ja tez jestem "ten wiek" i musze Ci przyznac ze sampooczucie podczas przyjmowania provironu jest o niebo lepsze niz braku tego specyfiku.
Co do prostaty i watroby sa przeciez oslonki(proscar oraz essentilae forte) zreszta wiesz o tym .
Dla mnie priorytetem jest zdrowie i zanim cos przyjmuje to sie dlugo zastanowie.
Jedynym bledem bylo uzycie arimidexu...

proscar nie ochroni prostaty przed provironem..
co do samopoczucie to wierze na słowo
ale wiesz, jak ktos chce miec wlosy do np 50r.z minimum to raczej lepiej rozwazyc co innego
takie moje zdanie.. ale wiadomo genetyka i indywidualna odpowiedz na srodki to podstawa do takich decyzji

Widze ze sama elita tu przesiaduje
Mam nadzieje ze moge sie dolaczyc

Bul - 250mg/week zrobi Ci dobrze - towar bedzie mial (szczegolnie u Ciebie) gdzie sie rozchulac. Co do nolvy/oxo w trakcie - przyjmowales m1t - wiec wiesz czy jestes podatny na estro czy nie .....A co do odbloku - ja proponuje z nolvy, nie sa to kosmiczne dawki, wiec nolva ( z malym frontem ) po cyklu powinna dobrze wypasc. to tylko IMO

Mam jeszcze pytanko do oma ( lub kogokolwiek innego kto przyjmowal 6oxo ) - jesli dziala niemal jak a-dex - to czy oprocz blokowania konwersji - blokuje tez przyrosty jak a-dex ?? bo jesli tak to srednio widze wlaczenie 6oxo do cyklu bulla
BTW. Om - odbierz priv

BTW blokerów 5ar - tak fogle to jest coś bardziej efektywniejszego niż finasteride, który sprawdza się w leczeniu bądź zapobieganiu hiperlazji gruczołu krokowego BPH czyli z angielskiego Benign Prostatic Hyperlasia - jest taki nowo opracowany środek - DUTASTERIDE, który działa w systemie dual - działa na zasadzie podwójnej (zdwojonej) efektywności - czyli jest to podówjny inhibitor 5ar typu I jak i II.



"Dutasteride
In spite of the promise of 5alpha-reductase inhibitors for benign prostatic hyperplasia (BPH), only one compound to date has reached the market: Merck & Co.'s finasteride (Proscar). However, as a selective type 1 isozyme inhibitor, finasteride has proved only moderately effective in treating BPH and more potent compounds are clearly needed. Dutasteride (GI-198745) is a dual inhibitor of 5alpha-reductase type 1 and 2 isozymes which appears to be suitable for use alone or in combination with alpha1-adrenoceptor antagonists for the treatment of BPH and associated symptoms. Glaxo Wellcome is conducting phase III trials of the compound, with plans to file for regulatory approval in the U.S. and Europe this year.

In a randomized, double-blind, parallel group 4-week study, dutasteride (40 mg loading dose then 0.1, 0.5, 2.5 and 2.5 mg/day) was shown to more potently suppress dihydrotestosterone (DHT) as compared to finasteride (5 mg/d) in 53 subjects with benign prostatic hyperplasia. DHT was dose-dependently suppressed in dutasteride-treated subjects with a maximum suppression of 95% occurring with the 5 mg dose; 0.5 mg, the lowest maximally effective dose, decreased DHT levels by 90% at 4 weeks and by 94% at 24 weeks, while finasteride only suppressed levels by 67 and 76%, respectively. Although testosterone levels increased (9-27%) with DHT suppression with both agents, levels were considered normal. Similar incidence of adverse effects was observed for placebo and both treatment groups although decreased libido was observed in subjects given 5 mg finasteride or dutasteride.

In an open-label, crossover study presented this summer to the American Urology Association, 38 healthy male subjects were given the alpha-blocker tamsulosin (0.4 mg/day) or terazosin (titrated up to 10 mg/day) for 14 days, followed by 7-day washout and subsequent treatment with dutasteride (0.5 mg/day following a 40-mg loading dose) for 21 days, followed finally by a 14-day treatment with the combination of dutasteride plus tamoxifen or terazosin. The results showed no significant drug interactions as regards pharmacokinetics. In addition, the incidence of adverse events of headache, dizziness, musculoskeletal pain, orthostasis, nausea and emesis was lower when dutasteride was coadministered with tamoxifen (18% vs. 29%) or terazosin (35% vs. 67%) compared to either drug alone.

Put this information about new treatments for Benign Prostatic Hyperplasia into perspective with the Prous Science's Drug R&D Backgrounders, a new series of internet-based, disease-focused reports. Follow this link (www.prous.com/backgrounders) to learn more about this valuable, continuously updated new research tool."

Było o tym już

Meta - bo kiedyś wkleiłem coś podobnego chyba.

ja pamietam jak Kazam jakies linki wklejal gdzie to mozna kupic

Ja wiem, kiedy topic się o tym pojawił - jak byłem nieobecny na forum - i czuwalem z ukrycia


Spoko jeden **** who brought it on!

Uriel nie wiedzialem o tym dzieki za info ,meta jakbys sie natknal na link Kazama gdzie ten wynalazek mozna kupic bede wdzieczny

"W praktyce Sustanon=Omnadren ... i poki co nikt mi nie udowodnil, ze jest inaczej "


W teori tez :

OMNADREN 250 = SUSTANON 250 formula wise at least.

OLD OMNADREN (prior of fall 2000)
Testosterone phenylproprionate 60 mg
Testosterone proprionate 30 mg
Testosterone isohexanoate 60 mg
Testosterone hexanoate 100mg

Caproate: Chemical Structure C6H12O2.
Also referred to as Hexanoic acid; hexanoate; n-Caproic Acid; n-Hexoic

acid; butylacetic acid; pentiformic acid; pentylformic acid; n-hexylic

acid; 1-pentanecarboxylic acid; hexoic acid; 1-hexanoic acid; Hexylic

acid; Caproic acid. This ester is identical to isocarpoate in terms of

atom count and weight, but is laid out slightly different (Isocaproate

has a split configuration, difficult to explain here but easy to see on

paper). Release duration would be very similar to isocaproate (levels

sustained for approximately one weak), perhaps coming slightly closer

to enanthate due to its straight chain. Caproate is the slowest

releasing ester used in Omnadren, which is why most athletes notice

more water retention with this compound.

NEW OMNADREN (after fall 2000)

Testosterone phenylproprionate 60 mg
Testosterone proprionate 30 mg
Testosterone isocaproate 60 mg
Testosterone caprinicum 100mg

Caproate (Caproic acid) is not the same as
Caprinicum (Capric acid) OK !!!!
Caprinicum = capric acid NOT Caproic acid
So Capronicum is different name for Deconate.

Decanoate: Chemical Structure C10H20O2.
Also referred to as decanoic acid; capric acid; caprinic acid; decylic

acid, Nonanecarboxylic acid. The Decanoate ester is most commonly used

with the hormone nandrolone (as in Deca-Durabolin) and is found in

virtually all corners of the world. Testosterone decanoate is also the

longest acting constituent in Sustanon, greatly extending its release

duration. The release time with Decanoate compounds is listed to be as

long as one month, although most recently we are finding that levels

seem to drop significantly after two weeks. To keep blood levels more

uniform, athletes (as they have always known to do) will follow a

weekly injection schedule.

THE ONLY REASON JELFA NAMES DECONATE AS CAPRONICUM (CAPRIC) DUE TO

PATENT FORMULA HELD BY ORGANON BUT GENETIC NAMES CAN NOT AND WILL NOT

BE PATENTED.